Recently I met with a wonderful woman, who shared with me her experience. Super fit and healthy this lady had a stroke in her 30's !! The cause of it was traced back to her time diving (specifically the clearing of the mask)
Not meaning to frighten anyone, but I have heard of this before and this lady told me of a 24 year old power lifter who suffered the same thing.
Breathing is our life force and when we hold our breath or cause inter cranial pressure (straining during exercise) this is a risk that is not often talked about.
Things to watch out for .. severe headaches that you havent had before ... one pupil becomes much larger than the other ..
many thanks M for passing this information on ...
Arterial dissection is a pathologic process characterized by splitting of the media or subadventitial layer of the arterial wall. The inside wall detaches causing a flap within the artery. Dissection of either the internal carotid artery or the vertebral artery was once considered uncommon.
However, in the past few decades, it has been increasingly recognized as a cause of a stroke in young adults. Dissection causes only 0.4% to 4% of all strokes in the general population[1,2] but causes 5% to 20% of strokes among young patients.[2-5
The pathophysiology of carotid artery dissection can be either traumatic or spontaneous. The true incidence of spontaneous dissection is unknown. Once considered rare, increased awareness, combined with noninvasive evaluation by ultrasonography and MRA, has shown a more frequent occurrence.[15]
Physical exertion or "trivial trauma" has been implicated as a triggering mechanism in cervicocephalic arterial dissection. [7] Carotid dissection had been reported after vomiting,[15] after scuba diving,[14,18] ceiling painting, nose blowing, during sporting activity without trauma,[22,23] after chiropractor manipulation,[19] after a visit to a hairdresser,[24] and after bronchoscopy.[25] Furthermore, few studies of animal models of carotid dissection are available. Kahler et al[26] described an internal carotid artery dissection model in a rabbit, though, unlike clinical situations, no local arterial or cerebral ischemic complications developed.
Based on our review, spontaneous internal carotid artery dissection is more common than most clinicians appreciate. However, even in the presence of existing signs and symptoms, the diagnosis can be missed by experienced physicians of all specialties.[27] The clinical picture varies from mild cerebral and/or cranial nerve dysfunction to a completed stroke
In 41% of patients, the interval between the first symptoms (local signs and/or transient ischemic attacks) and the onset of a stroke ranged from a few minutes to 31 days. VAD has been associated with a 30% mortality rate in the acute phase. Death is the result of extensive intracranial dissection, brainstem infarction, or subarachnoid hemorrhage.
Not meaning to frighten anyone, but I have heard of this before and this lady told me of a 24 year old power lifter who suffered the same thing.
Breathing is our life force and when we hold our breath or cause inter cranial pressure (straining during exercise) this is a risk that is not often talked about.
Things to watch out for .. severe headaches that you havent had before ... one pupil becomes much larger than the other ..
many thanks M for passing this information on ...
Arterial dissection is a pathologic process characterized by splitting of the media or subadventitial layer of the arterial wall. The inside wall detaches causing a flap within the artery. Dissection of either the internal carotid artery or the vertebral artery was once considered uncommon.
However, in the past few decades, it has been increasingly recognized as a cause of a stroke in young adults. Dissection causes only 0.4% to 4% of all strokes in the general population[1,2] but causes 5% to 20% of strokes among young patients.[2-5
The pathophysiology of carotid artery dissection can be either traumatic or spontaneous. The true incidence of spontaneous dissection is unknown. Once considered rare, increased awareness, combined with noninvasive evaluation by ultrasonography and MRA, has shown a more frequent occurrence.[15]
Physical exertion or "trivial trauma" has been implicated as a triggering mechanism in cervicocephalic arterial dissection. [7] Carotid dissection had been reported after vomiting,[15] after scuba diving,[14,18] ceiling painting, nose blowing, during sporting activity without trauma,[22,23] after chiropractor manipulation,[19] after a visit to a hairdresser,[24] and after bronchoscopy.[25] Furthermore, few studies of animal models of carotid dissection are available. Kahler et al[26] described an internal carotid artery dissection model in a rabbit, though, unlike clinical situations, no local arterial or cerebral ischemic complications developed.
Based on our review, spontaneous internal carotid artery dissection is more common than most clinicians appreciate. However, even in the presence of existing signs and symptoms, the diagnosis can be missed by experienced physicians of all specialties.[27] The clinical picture varies from mild cerebral and/or cranial nerve dysfunction to a completed stroke
In 41% of patients, the interval between the first symptoms (local signs and/or transient ischemic attacks) and the onset of a stroke ranged from a few minutes to 31 days. VAD has been associated with a 30% mortality rate in the acute phase. Death is the result of extensive intracranial dissection, brainstem infarction, or subarachnoid hemorrhage.
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